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In vitro data and mouse studies suggest that NE proteolytic activity triggers an inflammatory process that induces goblet cell metaplasia, which results in overexpression of mucins that contribute to the symptomatology of CRS (66, 67)

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A., Sparks, S

It indicated that PV2-S71 + PV3-N77 could indirectly inhibit tumor growth by blocking the PD-1/PD-L1 pathway and further promoting the infiltration of CD8 + T cells into tumor tissues

A new rat model of type 2 diabetes: the fat-fed, streptozotocin-treated rat